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- Dec 5, 2020
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And I actually believe that the figure from that small study, of 109g/m2, seems low. I would not be surprised if other studies find that those with severe AS have higher LV mass on average than that.the normal rate for men is up to 94
difference at the level of measurement error of average indicators
A few months ago a new member poste that his wall thickness was about 13mm and 17mm. The 17 figure is getting up there. You definitely do not want it to get that high, in my view, due to the risk that you will still have residual LVH after remodeling from AV surgery.
The study below found that 44% of patients had residual LVH after valve surgery, after a mean follow up time of 6 years. These were patients with aortic stenosis. The fact that 44% still had it after remodeling would suggest to me that the figure of 47% from the previous study linked was low. I think that most with severe AS have some degree of LVH.
"Residual LVH was present in 44% of patients after AVR"
Conclusions: "Residual LVH after AVR is common and is associated with worse prognosis, particularly in women."
https://pubmed.ncbi.nlm.nih.gov/26188338/
Some degree of remodeling does occur generally. Mine did remodel to completely normal wall thickness and mass, as I noted previously. Soon after I was diagnosed with LVH, I had a consult with a cardiologist at Scripps and I did bring up my concerns about LVH. He said that mine was still in the range in which I was likely to get complete remodeling, before fibrosis occurs. He pulled up some images with several stages of LVH. I think that there were 5 or 6. I can't recall if I was at the first or second stage, but he indicated that I was still a couple of stages away from the point at which fibrosis was likely to occur. As I understood it, once fibrosis occurs, the LV will not completely remodel and there will be residual LVH typically.
Additionally, the finding in that study that 44% of AS patients still had residual LVH 6 years after valve surgery suggests that many are not getting surgery done in time to prevent permanent damage to the left ventricle.
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